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Scientific Researches On:

Betulinic Acid (Birch Bark Extract)

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Betulinic Acid-Induced Apoptosis in Glioma Cells: A Sequential
Requirement for New Protein Synthesis, Formation of Reactive
Oxygen Species, and Caspase Processing

Betulinic Acid-induced Programmed Cell Death in Human
Melanoma Cells Involves Mitogen-activated Protein
Kinase Activation

Betulinic Acid Triggers CD95 (APO-lIFas)- and p53-independent Apoptosis via Activation of Caspases in Neuroectodermal Tumors I

Betulinic Acid Triggers CD95 (Apol/Fas)- and p53-independent Apoptosis viaActivation of Caspases in Neuroectodermal Tumors II

Activation of Mitochondria and Release of Mitochondrial
Apoptogenic Factors by Betulinic Acid

Effects of Betulinic Acid Alone and in Combination with Irradiation in Human Melanoma Cells

Betulinic Acid-induced Mcl-1 Expression in Human Melanoma –
Mode of Action and Functional Significance

Betulinic Acid Suppresses Carcinogen-Induced NF- B Activation
Through Inhibition of I B Kinase and p65 Phosphorylation:
Abrogation of Cyclooxygenase-2 and Matrix Metalloprotease-9

 

Biotransformation of the Antimelanoma Agent Betulinic Acid
by Bacillus megaterium ATCC 13368

Betulinic Acid Inhibits Prostate Cancer Growth through Inhibition
of Specificity Protein Transcription Factors

Mitochondrion as a Novel Target of Anticancer
Chemotherapy

A Key Role for Caspase-2 and Caspase-3 in the Apoptosis
Induced by 2-Chloro-2 -deoxy-adenosine (Cladribine) and 2-
Chloro-adenosine in Human Astrocytoma Cells

Molecular Ordering of Apoptosis Induced by Anticancer Drugs in
Neuroblastoma Cells

The Bax/Bcl-2 Ratio Determines the Susceptibility of Human
Melanoma Cells to CD95/Fas-Mediated Apoptosis

Apoptosis Protease Activator Protein-1 Expression Is Dispensable for Response of Human Melanoma Cells to Distinct Proapoptotic Agents

Resistance of Human Astrocytoma Cells to Apoptosis
Induced by Mitochondria-Damaging Agents: Possible
Implications for Anticancer Therapy

Luteolin, an emerging anti-cancer flavonoid, poisons eukaryotic
DNA topoisomerase I

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